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Obesity, Glucose and Insulin
Chronic Dysglycemia, Reduced Thermogenesis, Fat Production and Insulin Resistance
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Obesity, Glucose & Insulin

Weight Gain and Metabolic Dysglycemia

Obesity is often triggered and perpetuated by metabolic and hormonal imbalances that create a vicious cycle of chronic dysglycemia, reduced thermogenesis, and altered fat production. This type of chronic metabolic dysregulation can make it very difficult to lose weight effectively without first addressing key functional imbalances.

Glucose and insulin are primary players in the etiology of obesity. The balance between glucose and insulin in the blood can influence levels of leptin, the "hunger hormone" believed to play a strong role in regulating human appetite and metabolism. Insulin is released in response to rising blood glucose, and transports glucose to cells, where it can be used for fat production. So it comes as no surprise that upper body fat and abdominal obesity are strongly associated with "insulin resistance," a condition that arises when cell receptors resist insulin's attempt to deposit glucose, resulting in hyperinsulinemia, a state of chronically high levels of insulin. In fact, some researchers have suggested that consistently high insulin levels, both fasting and after eating, may actually instigate the metabolic process associated with obesity by increasing lipid levels in the blood.

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Obesity and Weight Loss
Obesity is now the "trillion dollar disease". This reflects the social costs of obesity, in health and lost production, as well as the weight loss drug profits that might be made for any pharmaceutical company that finds a drug-cure for obesity. The search for viable weight loss medications continues, although pharmaceutical weight loss products still have significant side effects.

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